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Approaching and Hitting the Male Wall
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<blockquote data-quote="thoughtgypsy" data-source="post: 948518" data-attributes="member: 571"><p>Fasting also mimics the effects of Calorie Restriction (CR) through the process of autophagy by the mTOR (cell growth) and AMPK (cell energy) pathways. Most of the research being done on this topic is conducted on simple organisms like C. elegans or mice, though what has been thus far is promising. Primate studies are suggesting clinical significance in humans.</p><p></p><p>The mitochondrial theory of aging suggests that free radicals (reactive oxygen species) can attach to molecules in the body and introduce cell replication errors over time. By reducing the mTOR activity (cell replication rate), the accumulation of errors is slowed.</p><p><a href="https://en.wikipedia.org/wiki/Mechanistic_target_of_rapamycin#Aging" target="_blank">https://en.wikipedia.org/wiki/Mechanistic_target_of_rapamycin#Aging</a></p><p></p><p>Lipolysis (through the AMPK pathway) may extend life by slowing the generation of reactive oxygen species, which are a by-product of cellular energy (ATP) production. This may make the clearance of reactive oxygen species from the body easier. Increased AMPK activity is associated with longer life span in other species.</p><p><a href="http://roguehealthandfitness.com/activating-ampk-lifespan-extension/" target="_blank">http://roguehealthandfitness.com/activating-ampk-lifespan-extension/</a></p><p><a href="https://en.wikipedia.org/wiki/AMP-activated_protein_kinase#Maximum_life_span" target="_blank">https://en.wikipedia.org/wiki/AMP-activated_protein_kinase#Maximum_life_span</a></p><p><a href="https://en.wikipedia.org/wiki/Hormesis#Mitohormesis" target="_blank">https://en.wikipedia.org/wiki/Hormesis#Mitohormesis</a></p><p></p><p>Intermittent fasting stimulates the same lifespan-increasing activity as CR, but is much more convenient. By restricting the feeding window to 8 hours or less in a day, someone can still get meet their caloric needs while gaining the same benefits of CR.</p><p></p><p>A discussion on the timing of IF for those who remain active: <a href="http://roguehealthandfitness.com/an-intermittent-fasting-schedule/" target="_blank">http://roguehealthandfitness.com/an-intermittent-fasting-schedule/</a></p><p>Further discussion on anti-aging strategies: <a href="http://roguehealthandfitness.com/the-optimal-anti-aging-strategy/" target="_blank">http://roguehealthandfitness.com/the-optimal-anti-aging-strategy/</a></p><p></p><p>Perhaps I am wrong, but I'm of the belief that peak performance and longevity are not directly correlated. Our bodies are machines, and when pushed beyond the point of exhaustion, damage can be incurred. Endurance athletes have more efficient hearts (lower resting heartrates) due to the necessity to deliver large volumes of oxygen in short amounts of time. But there is a point where stress is excessive and damage occurs. It is difficult to know where this upper bound is, but there are some studies out there that may give a better idea.</p><p><a href="http://www.ncbi.nlm.nih.gov/pmc/articles/PMC3538475/" target="_blank">http://www.ncbi.nlm.nih.gov/pmc/articles/PMC3538475/</a></p><p></p><p>Weightlifting seems to be the most efficient method for the body to perform work without inducing cardiovascular damage. It stimulates testosterone production and prevents muscle loss (a reliable indicator of premature death) through the inhibition of myostatin. Exercise activity is a very reliable indicator of longevity: <a href="http://roguehealthandfitness.com/exercise-capacity-powerfully-predicts-survival/" target="_blank">http://roguehealthandfitness.com/exercise-capacity-powerfully-predicts-survival/</a></p><p></p><p></p><p></p><p>Not sure if these would be directly related to hGH production, but these are other things I practice:</p><p></p><p>- Eating carbohydrates <em>following</em> intense workouts (to replace muscle glycogen expended during training)</p><p>- Eating probiotics (sauerkraut, kimchi, yogurt, or pills) to extract maximum calories from digested food (less fat deposition).</p><p>- Eating/drinking polyphenols (green tea or EGCG, coffee, blueberries, grapes[red wine]) to help clear reactive oxygen species. Controversial: <a href="https://www.ncbi.nlm.nih.gov/pubmed/17327526" target="_blank">https://www.ncbi.nlm.nih.gov/pubmed/17327526</a></p><p>- Restricting fat content during carbohydrate-laden meals (fat is calorically dense, carbs are glycemically "fast"; combining them raises the volume of fat deposition)</p><p>- Sauteing veggies or combining them with fatty food (some vitamins are fat soluble)</p><p>- Avoiding dairy, even from grass fed cows (rBGH and BGH are given to dairy cows to produce excessive milk production; but they also exist naturally in high levels in cow's milk even before human intervention)</p><p>- Getting a source of omega-3 fatty acids (eggs, fish or fish oil, avocados, flaxseed, walnuts) to reduce inflammation</p></blockquote><p></p>
[QUOTE="thoughtgypsy, post: 948518, member: 571"] Fasting also mimics the effects of Calorie Restriction (CR) through the process of autophagy by the mTOR (cell growth) and AMPK (cell energy) pathways. Most of the research being done on this topic is conducted on simple organisms like C. elegans or mice, though what has been thus far is promising. Primate studies are suggesting clinical significance in humans. The mitochondrial theory of aging suggests that free radicals (reactive oxygen species) can attach to molecules in the body and introduce cell replication errors over time. By reducing the mTOR activity (cell replication rate), the accumulation of errors is slowed. [URL]https://en.wikipedia.org/wiki/Mechanistic_target_of_rapamycin#Aging[/URL] Lipolysis (through the AMPK pathway) may extend life by slowing the generation of reactive oxygen species, which are a by-product of cellular energy (ATP) production. This may make the clearance of reactive oxygen species from the body easier. Increased AMPK activity is associated with longer life span in other species. [URL]http://roguehealthandfitness.com/activating-ampk-lifespan-extension/[/URL] [URL]https://en.wikipedia.org/wiki/AMP-activated_protein_kinase#Maximum_life_span[/URL] [URL]https://en.wikipedia.org/wiki/Hormesis#Mitohormesis[/URL] Intermittent fasting stimulates the same lifespan-increasing activity as CR, but is much more convenient. By restricting the feeding window to 8 hours or less in a day, someone can still get meet their caloric needs while gaining the same benefits of CR. A discussion on the timing of IF for those who remain active: [URL]http://roguehealthandfitness.com/an-intermittent-fasting-schedule/[/URL] Further discussion on anti-aging strategies: [URL]http://roguehealthandfitness.com/the-optimal-anti-aging-strategy/[/URL] Perhaps I am wrong, but I'm of the belief that peak performance and longevity are not directly correlated. Our bodies are machines, and when pushed beyond the point of exhaustion, damage can be incurred. Endurance athletes have more efficient hearts (lower resting heartrates) due to the necessity to deliver large volumes of oxygen in short amounts of time. But there is a point where stress is excessive and damage occurs. It is difficult to know where this upper bound is, but there are some studies out there that may give a better idea. [URL]http://www.ncbi.nlm.nih.gov/pmc/articles/PMC3538475/[/URL] Weightlifting seems to be the most efficient method for the body to perform work without inducing cardiovascular damage. It stimulates testosterone production and prevents muscle loss (a reliable indicator of premature death) through the inhibition of myostatin. Exercise activity is a very reliable indicator of longevity: [URL]http://roguehealthandfitness.com/exercise-capacity-powerfully-predicts-survival/[/URL] Not sure if these would be directly related to hGH production, but these are other things I practice: - Eating carbohydrates [i]following[/i] intense workouts (to replace muscle glycogen expended during training) - Eating probiotics (sauerkraut, kimchi, yogurt, or pills) to extract maximum calories from digested food (less fat deposition). - Eating/drinking polyphenols (green tea or EGCG, coffee, blueberries, grapes[red wine]) to help clear reactive oxygen species. Controversial: [URL]https://www.ncbi.nlm.nih.gov/pubmed/17327526[/URL] - Restricting fat content during carbohydrate-laden meals (fat is calorically dense, carbs are glycemically "fast"; combining them raises the volume of fat deposition) - Sauteing veggies or combining them with fatty food (some vitamins are fat soluble) - Avoiding dairy, even from grass fed cows (rBGH and BGH are given to dairy cows to produce excessive milk production; but they also exist naturally in high levels in cow's milk even before human intervention) - Getting a source of omega-3 fatty acids (eggs, fish or fish oil, avocados, flaxseed, walnuts) to reduce inflammation [/QUOTE]
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